Lyme_protocol_Jan06.pdf

Lyme disease: A Look Beyond Antibiotics

Dietrich K.Klinghardt, MD, PhD

Bellevue WA

aant@neuraltherapy.com 1/7/05 and 1/7/06

425-637-9339

In the last decade the majority of outcome-oriented physicians observed a

major shift: we realized that it was neither the lack of vitamins or growth

hormone that made our patients ill. We discovered that toxicity and chronic

infections were most often at the core of the client’s suffering. We watched

the discussion, which infection may be the primary one: mycoplasma, stealth

viruses, HHV-6, trichomonas, Chlamydia pneumoniae, leptospirosis,

mutated strep, or what else?

The new kid on the block is Borrelia burgdorferi (Bb) and some of us have

looked at it for a long time as possibly being the bug that opens the door for

all the other infections to enter the system. Lyme disease has become a

buzzword in the alternative medical field. Since none of the recommended

treatments are specific to either one of the microbes, we can never assume

that we really know what we treated once a patient has recovered.

Microbiologist Gitte Jensen, PhD had shown, that the older we get, the more

foreign DNA is attached to our own DNA. Somewhere along the line

pathogenic microbes invade the host’s DNA and become a permanent part of

it. Since we use only 2% of our DNA, it may not be a problem. In fact, it

may make us who we finally become. It may also cause a number of

symptoms and chronic illness. Genius Guenther Enderlein’s discoveries take

us off the hook: if one microbe can change into another given the right

environment, why bother to find out, who we are infected with? The book

“Lab 257” suggests that Bb is an escaped man-made US military bio-warfare

organism (just like myoplasma incognitus and HHV 6).

Other authors suggest that different subtypes of Borrelia, which cause illness

in humans, such as B. afzelii and B.garinii have probably existed longer then

B.burgdorferi and occur naturally (1, 2) and have been with us for a long

time, maybe centuries or much longer then that.

Neurologist Prof. J. Faust MD, PhD of the Albert-Ludwig University in

Freiburg, Germany (3) related many neurological and psychiatric illnesses to

spirochete infections as early as the 1960s. He was so skilled in his clinical

knowledge that he could – based on clinical neurological symptoms –

accurately predict which valley in the Black Forest the infected patient was

from! This clearly was a time before Bb - showing that non-syphilis

spirochete infections were around earlier then the famous Bb outbreak in

Connecticut in the mid seventies. It also makes a strong statement to the fact

how easily these creatures may mutate and adapt to local conditions. It may

however validate the findings published in “Lab 257”: Tuebingen, the place

where German/US warfare spirochete expert Traub was continuing his

spirochete experiments in the early 50s, is situated in the Black Forest also.

Were these spirochetes genuine or have they escaped from a university

laboratory?

Making the diagnosis

It appears that many patients with MS, ALS, Parkinson’s disease, autism,

joint arthritis, chronic fatigue, sarcoidosis and even cancer are infected with

Borrelia burgdorferi. But is the infection causing the illness or is it an

opportunistic infection simply occurring in people weakened by other

illnesses.

My experience is based on:

a) using direct microscopic proof of the presence of Borrelia burgdoferi

(Bb) and other spirochetes (4, 5)

b) the information many affected clients have brought to me

c) my own clinical training and experience (30 years in Medical practice,

15 years Bb cognizant)

d) ART testing (autonomic response testing), which is the most advanced

and scientifically validated method of muscle testing (6)

e) regular lab parameters affected by Lyme:

•

Abnormal lipid profile (moderate cholesterol elevation with

significant LDL elevation)

insulin resistance

•

borderline low wbc, normal SED rate and CRP

•

normal thyroid hormone tests but positive Barnes test and excellent

response to giving T3

•

type 2 (high cortisol, low DHEA) or type 3 adrenal failure (low

cortisol and DHEA)

low testosterone and DHEA

•

decreased urine concentration (low specific gravity)

•

complex changes in cytokines, interferones, NK cells, white blood

cell indicators, etc.

Bb tends to infect the B-lymphocytes and other components of the immune

system which are responsible for creating the antibodies, which are then

measured by an ELISA test or Western Blot test. Since antibody production

is greatly compromised in infected individuals, it makes no sense to use

these tests as the gold standard or benchmark for the presence of Bb (7). We

also are aware that in endemic areas in the US up to 22% of stinging flies

and mosquitoes (2, 8, 9, and 10) are carriers of Bb and co-infections. In

South East Germany and Eastern Europe 12 % of mosquitoes have been

shown to be infected. Also many spiders, flees, lice and other stinging

insects carry spirochetes and co-infections.

Making the history of a tick bite a condition for a physician to be willing to

even consider the possibility of a Bb infection seems cynical and cruel.

To use conventional diagnostic tests such as the Western Blot, one has to

think in paradoxes: the patient has to be treated with an effective treatment

modality first before the patient recovers enough to produce the antibodies,

which then are looked for in the test. A positive Western Blot proves that the

treatment given worked to some degree. A negative Western Blot does not

and cannot prove the absence of the infection.

Having taken another route altogether, we have recognized that today many

if not most Americans are carriers of the infection. Most infected people are

symptomatic, but the severity and type of the symptoms varies greatly. The

microbes often invade tissues that had been injured: your chronic neck pain

or sciatica really may be a Bb infection. The same may be true for your

chronic TMJ problem, your adrenal fatigue, your thyroid dysfunction, your

GERD and many other seemingly unrelated symptoms. Many Bb symptoms

are mistaken for problems of natural or premature aging.

In most places the diagnosis of an active Bb infection is made only if the

symptoms are severe, persistent, obvious, and many non-specific and

fruitless avenues of treatment have been exhausted. Acute new “typical”

cases of Bb infection are rare in my practice. Symptoms tend to get stranger

and more obscure every year.

Frequently, if the patient is fortunate enough to see a practitioner who is

“Lyme cognizant”, the diagnosis of a supposedly fresh case of symptomatic

Lyme disease is made when a significant tissue toxin level has been reached

(threshold phenomenon) or when a new co-infection has occurred recently.

The symptoms can mimic any other existing medical, psychological or

psychiatric condition. The list of significant co-infections is limited:

roundworms, tapeworms, threadworms, toxoplasmosis, giardia and amoebas,

clostridia, the herpes virus family, parvovirus B 19, active measles (in the

small intestine), leptospirosis, chronic strep infections and their mutations,

Babesia, Brucella, Ehrlichiosis, Bartonella, mycoplasma, Rickettsia,

Bartonella and a few others. Molds and fungi are always part of the picture.

The pattern of co-infections and the other preexisting conditions such as

mercury toxicity determine the symptom-picture but not the severity.

The severity of symptoms correlates most closely with the overall

summation or body burden of coexisting conditions and with the genetically

determined ability to excrete neurotoxins. The genes coding for the

glutathione S-transferase and for the different alleles of apolipoprotein E

(E2, E3 and E4) play a major role. E2 can carry twice as much sulfhydryl-

affinitive toxins (such as mercury and lead) out of the cell as the E3 subtype,

E4 carries out none. Trouble in the methylation, acetylation and sulfation

pathways is also common. Other factors, such as diet and food allergies, past

toxic and electromagnetic exposures, emotional factors and unhealed

ancestral trauma, scar interference fields and occlusal jaw and bite problems

are also important (6). The severity of symptoms is not related to the number

of spirochetes in the system but rather to the individual’s immune responses.

Taken all of the above into account, we do not distinguish between people

who have the Bb infection and those who don’t. We distinguish between

people who have Lyme disease and those who don’t.

a) patients who are infected with any type of Borrelia and are

symptomatic have “Lyme” disease

b) healthy people who are not symptomatic often already have a

spirochete infection as well. They may or may not be disasters

waiting to happen. But they do not (yet) have Lyme “disease”.

Most often several of the “co-infections” are already present prior to the

infection with Bb or other spirochetes.

In treatment we focus on exploring the difference between symptomatic and

asymptomatic carriers. We treat what the symptomatic person is missing

(such as enough magnesium in the diet) or has extra (such as mercury)

compared to the asymptomatic one.

The group suffering most is newborn babies and young children, who rarely

are diagnosed correctly and therefore are not treated appropriately. They

often carry the labels ADHD, autistic spectrum disorder (ASD), seizure

disorder and others. Detoxifying these kids with transdermal DMPS and

treating the chronic infections is often curative.

The 3 Components of Lyme disease

Lyme disease has three components, which should be recognized and

addressed with treatment:

Component #1: The presence of spirochete infection and co-infections

The co-infections are bacterial, viral, fungal and parasitic. Since the

spirochetes paralyze multiple aspects of the immune system, the organism is

without defenses against many microbes. Many - if not most - of the co-

infections are really a consequence of the spirochete infection and not truly a

simultaneously occurring “co-infection”.

For this aspect of treatment we use pulsed electromagnetic fields (KMT-

microbial inhibition frequencies), niacin in high doses (12) herbs, minerals,

bee venom (6) and - sometimes - ant parasitic medication and antibiotics.

The KMT microcurrent technology is new and revolutionary (17). The

instruments are FDA approved for pain control. Designed by Japanese

engineers they use four different - but simultaneously applied - high

frequency superimposed biological waveforms. The interference pattern is

creating thousands of harmonics which are then manipulated into the

specific published microbial inhibition frequencies (against Bb, mycoplasma

etc.).

This stealthy microcurrent travels freely through the body reaching every

tissue. The instrument measures the skin conductance over a 100

times/second adjusting the amperage constantly (so that the body never

creates habituation/resistance against it). The microbes are inhibited in

their metabolic and sexual activity and gradually die out or disappear from

the body

The instrument looks not much different then a TENS unit and is applied via

four electrodes to the skin or used by translating the electric field into a

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